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On the other hand, macrophage/-cell crosstalk can drive -cell dysfunction and apoptosis when physiological equilibrium is disturbed by the inflammatory signals present during the development of T1D and T2D (16) ( Figure 3 )
2025;11:e41188
Human iPSCs derived astrocytes rescue rotenone-induced mitochondrial dysfunction and dopaminergic neurodegeneration in vitro by donating functional mitochondria
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Kava for Generalized Anxiety Disorder: A Review of Current Evidence
Free radicals are unstable molecules that can damage cells and contribute to aging, inflammation, and chronic disease
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